The AIDS epidemic, caused by a mysterious monkey immunosuppressive virus that suddenly crossed into humans and caused an epidemic of cancer, did not catch the entire medical community off guard. Quite the contrary. For decades prior to the AIDS outbreak, cancer virus researchers had been creating, manipulating, studying and testing immunosuppressive viruses with selective immune system damaging and cancer-causing properties remarkably similar to AIDS. Moreover, several of these immunosuppressive viruses (including those from monkeys and humans) were being grown in human cell cultures shortly before the AIDS epidemic broke out in humans.
These unique viruses were created as part of the U.S. government’s war on cancer in a crash program to develop vaccines. These experimental immunosuppressive viruses, which were isolated from mice, cats, cows and monkeys in the 1960s and 1970s, were successfully used to destructively manipulate the immune systems of a variety of lab animals as a means of controllably increasing susceptibility to cancer. This research was conducted so that scientists could learn more about precisely how immune system dysfunction contributed to cancer growth. The innovative research with these novel immunosuppressive viruses provided valuable information on the role that various components of the immune system played in cancer prevention and aided the development of various means of manipulating the immune system to prevent cancer in animals.
Interestingly, the selective immune system damage caused by the AIDS epidemic in humans is supplying cancer researchers with invaluable data eerily similar to that which they obtained in the 1970s by deliberately injecting the immunosuppressive viruses they created in animal test subjects. This is so because the cancer epidemic initiated by AIDS is providing observant researchers a wealth of invaluable data about how cancer grows, from its earliest to its latest phases, in human subjects with selectively impaired immune systems. By constructing huge databases on how cancer grows in AIDS patients as their immune systems systematically degrade, researchers are quietly finding such powerful clues as to the mechanisms behind the increased susceptibility to cancer in human AIDS patients, especially those cancers thought to be caused by viruses...that many are now confidently predicting the imminent development of human cancer vaccines. Such a phenomenal development would mirror similar progress made earlier in animal cancer research with immunosuppressive and cancer viruses.
It is at this point that a terrible question must be asked: Could HIV, and the epidemic of human cancers and death resulting from it, be the result of a sinister cross-species experiment with a monkey cancer virus designed as a means of intentionally inducing cancer in human subjects as a research and diagnostic tool in the development of human cancer vaccines? If so, was such an experiment modeled after decades of similar animal experiments in which well-characterized immunosuppressive viruses similar to HIV were injected in test subjects so that cancer researchers could monitor the progress of induced cancer growth as the animals’ immune systems deteriorated? And was this why researchers were growing monkey and human immunosuppressive viruses in human cell cultures, determining the pathogenic effects on T-cells, shortly before HIV began to destroy T-cells in the immune systems of human populations?
Unfortunately, there are numerous disturbing precedents indicating that cancer researchers would do this very thing. These precedents supply deeply disquieting evidence consistent with the terrifying hypothesis that the AIDS epidemic, caused by a simian virus that has resulted in an unprecedented international human cancer epidemic, is the result of the wider application of shockingly unethical and dangerous human cancer experiments with simian cancer viruses conducted throughout the 1950s and 60s.
As will be discussed in greater detail in a later section, in one set of these cross-species cancer vaccine experiments, immunosuppressed rats and humans were given the same set of cancer-inducing virus injections so that the tumors that developed in each species could be systematically compared. In a similar set of such immunosuppressive experiments, tumors were deliberately induced in human subjects as a result of injections with monkey tumor and cancer viruses, so that researchers could compare the resultant human tumors with those that grew in the monkeys. As an extension of this type of work, in the 1970s, shortly before AIDS began in human populations, researchers began combining primate cancer viruses that had been injected in human subjects with primate immunodeficiency viruses in human cell cultures.
For example, while the public is completely ignorant of this fact, simian immunosuppressive viruses (SIVs) such as the Mason Pfizer Monkey Virus, one of 3 types of SIV, were available to researchers as early as 1970, when the virus was grown in cell cultures. Well before AIDS, the Mason Pfizer Monkey Virus (MPMV) was shown to induce immunodeficiency states in monkeys in the early 1970s, as well as the 1980s. Even more provocative is the fact that cancer researchers were coaxing this immunosuppressive monkey virus to grow in human cells in the late 1970s, just before HIV broke out in human populations and caused an epidemic of sarcoma. In these experiments, MPMV was mixed with SV40, a monkey cancer virus (which had already been injected in humans to cause cancer) and the Rous sarcoma virus in human cell cultures. Since HIV is thought to be caused by an SIV, it is amazing that these intriguing pre-AIDS experiments have not been publicized.
The national security establishment, which worked closely with the viral cancer research community, is likewise not a disinterested party in the AIDS epidemic. While cancer researchers are benefiting from the selective depopulation of certain cells in the immune systems of HIV victims, it could be argued that the national security planners in fact are benefiting from the resultant selective destruction of human populations. This depopulation effect is fulfilling national security goals for the Third World, where 90% of AIDS deaths occur and where 95% of world population growth occurs, that were outlined in classified national security planning documents shortly before AIDS broke out.
The latter section of this study will examine the hypothesized linkage between cancer research and the interests of the national security community. Specifically, evidence will be presented to support the thesis that the AIDS epidemic was deliberately started as an exercise in biowarfare conducted under the cover of a human cancer experiment. This would explain why the HIV epidemic is selectively depopulating regions of the globe that the national security establishment had targeted for depopulation in the 1970s as a means of maintaining access to resources in the developing world. Additionally, the "cancer vaccine experiment" theory of the origin of AIDS could also explain not only why the AIDS virus selectively depopulates the exact components of the immune system that cancer researchers had been targeting for decades prior to AIDS but why homosexual populations are disproportionately infected by HIV and rare cancers. And finally, there is credible evidence that AIDS vaccine research vehicles have a linkage to the Gulf War Syndrome.